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The Influence of Tobacco Powerwall Advertising on Children
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Second-Hand Smoke

Introduction

As it became harder and harder to deny that first-hand smoke is a major killer, tobacco companies gradually shifted to a new strategy: blame smokers for the health effects of tobacco products. Deny the reality of addiction, and claim that smokers freely choose to expose themselves to the numerous diseases caused by tobacco smoke.

Whatever the ethics of advancing this argument, it has been an effective PR strategy. But it has been no help at all in dealing with the uncomfortable reality that second-hand smoke can, and often does, kill non-smokers. The industry’s only available line of defence is to contest the science, to claim that diluted poison is no poison at all.

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In 1993, the U.S. EPA published a major report on the effects of second-hand smoke, based on a thorough review of all the scientific literature. The agency went on to classify second-hand tobacco smoke as a Group A human carcinogen. In response, the tobacco industry launched a major campaign to discredit the environmental regulator, through the media, political lobbyists and the courts.

In July 1998, a U.S. District Court judge in North Carolina ruled that the EPA had failed to follow scientific procedure in reaching its conclusions about the link between cancer and second-hand smoke.

The decision is under appeal. Since the original report was published, several other panels of scientists in other countries have come to the same conclusions: second-hand smoke is a killer.

Executive Summary

The Consultation concluded that ETS is a real and substantial threat to child health, causing death and suffering throughout the world. ETS exposure causes a wide variety of adverse health effects in children, including lower respiratory tract infections such as pneumonia and bronchitis, coughing and wheezing, worsening of asthma, and middle ear disease. Childrens' exposure to environmental tobacco smoke may also contribute to cardiovascular disease in adulthood and to neurobehavioural impairment.

The Consultation also concluded that maternal smoking during pregnancy is a major cause of sudden infant death syndrome (SIDS) and other well-documented health effects, including reduced birth weight and decreased lung function. In addition, the Consultation noted that ETS exposure among nonsmoking pregnant women can cause a decrease in birth weight and that infant exposure to ETS may contribute to the risk of SIDS.

Conclusions:

Exposure to environmental tobacco smoke is a cause of lung cancer and, in those with long term exposure, the increased risk is in the order of 20-30%.

Exposure to environmental tobacco smoke is a cause of ischaemic heart disease and, if current published estimates of magnitude of relative risk are validated, such exposure represents a substantial public health hazard.

Smoking in the presence of infants and children is a cause of serious respiratory illness and asthmatic attacks.

Sudden infant death syndrome, the main cause of post-neonatal death in the first year of life, is associated with exposure to environmental tobacco smoke. The association is judged to be one of cause and effect.

Middle ear disease in children is linked with parental smoking and this association is likely to be causal.

The long and the short of scientific evidence about the negative effects of second-hand smoke. In November 1999, this report was republished as a monograph of the U.S. National Cancer Institute (see Monograph 10).

Another overview of the scientific evidence on second-hand smoke.


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According to the latest results from the Canadian Tobacco Use Monitoring Survey (CTUMS), for data collected between February and December 2005, slightly fewer than 5 million people, representing 19% of the population aged 15 years and older, were current smokers, of which 15% reported smoking daily. Approximately 22% of men were current smokers, higher than the proportion of women (16%).
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